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[5] Mechanisms [edit] For GPCRs generally, each mechanism of homologous desensitization begins with receptor phosphorylation by an associated G protein-coupled receptor kinase (GRK)

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Chronic hyperglycemia induces a proinflammatory state characterized by elevated levels of cytokines such as tumor necrosis factor-alpha (TNF-α), interleukin(IL)-1 beta, and IL-6