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Following the therapy was a consistent counteraction of early and full multiorgan failure (and heart failure, in particular) and thrombosis of the described occlusion/occlusion-like syndrome [66,71,72,73,74,75,76,77,78,79,80,81,82], peripherally and centrally, and thereby, counteraction of the later isoprenaline-infarction and isoprenaline-reinfarction [79]

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9,13 Other mechanisms of hepatotoxicity include the formation of toxic free radicals, such as peroxynitrite, from the reaction of superoxide and nitric oxide, subsequently forming nitrotyrosine adducts inside the mitochondria